For informational and research purposes only. Not medical advice. Content is aggregated from public sources. Always consult a qualified healthcare provider.
Performance & Growth Hormone · Comparison

ACE-031 vs Ghrelin

A neutral, side-by-side comparison of two performance & growth hormone peptides — their mechanisms, dosing ranges, administration routes, side effects, and research evidence. For research and educational purposes only.

ACE-031Ghrelin
CategoryPerformance & Growth HormonePerformance & Growth Hormone
Also known asACVR2B-Fc, ActRIIB-Fc fusion proteinGHRL, growth hormone-releasing peptide endogenous, ghrelinergic peptide, acyl ghrelin
EvidenceInvestigational (in trials)Investigational (in trials)
Dosing range0.3mg/kg–3mg/kg mg/kg, every 2–4 weeks (subcutaneous injection)0.5mcg/kg–2mcg/kg mcg/kg, IV or SC bolus; endogenous levels peak pre-meal
AdministrationSubcutaneous injectionIntravenous (research), Subcutaneous injection
Key side effectsNosebleeds (epistaxis — most common reported AE), Telangiectasia (visible small blood vessel dilation), Gum bleeding, Elevated hemoglobinAppetite stimulation, Transient hyperglycemia, Water retention, GH pulse stimulation
Cited sources2 references2 references

Key differences

  • Administration: ACE-031 — Subcutaneous injection; Ghrelin — Intravenous (research), Subcutaneous injection.
  • Dosing units differ: ACE-031 is dosed in mg/kg, Ghrelin in mcg/kg — they operate at different scales.
  • Frequency: ACE-031 is typically every 2–4 weeks (subcutaneous injection); Ghrelin is IV or SC bolus; endogenous levels peak pre-meal.

How each works

ACE-031

ACE-031 functions as a decoy receptor, binding myostatin, activin A/B, GDF-11, and BMP-9 in circulation before they can activate cellular ActRIIB receptors. In the Duchenne muscular dystrophy Phase 2 trial, ACE-031 produced meaningful lean body mass increases but was halted due to vascular side effects (epistaxis, telangiectasia) requiring dose and safety optimization.

Ghrelin

Ghrelin was discovered in 1999 as the endogenous ligand for the GHS-R1a receptor. Its acylated form activates GH release from the pituitary, stimulates appetite via NPY/AgRP hypothalamic neurons, and modulates energy homeostasis and fat storage. The des-acyl form (majority of circulating ghrelin) lacks GHS-R1a activity but has independent cardiovascular and cellular effects.

Read the full ACE-031 profileRead the full Ghrelin profile

This comparison aggregates public research and structured profile data for informational purposes only. It is not medical advice and does not recommend either compound for human use. Consult a qualified healthcare provider before considering any peptide.